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Antibacterial activities of the synthesized AgNPs at 50-150 ug/ml concentrations are shown in Fig. 6. The particles demonstrated remarkable antibacterial effects against the clinical bacterial strains of  mm for biosynthesized AgNPs using leaf strains. Though, the exact mechanism of  P  aerugin osa, S. aureus, S. Typhi and E. coli by inducing 10-20 nm zone of inhibi test organ sms (Table 1). These results corroborate the antibacterial activities of AgNPs reported in the previous studies (Priyadarshini et al., 2013; Shankar et al., 2014; Salem et al., 2014; Augustine et al., 2014; Lateef et al., 2015a, b; 2016d). For instance, most recently Lateef reported that the cocoa pod husk extract-mediated AgNPs induced zone of inhibition of 10-14 mm against clinical isolates of bacteria while Sale  et al. (2016a  m et al. (2014) reported zones of inhibition of 7-19 atex of Ficus sycomorus against some bacteri ity of AgNPs is not yet  isms. For instance, the a  and ibacte researchers have suggested many possible eff  rial acti known but  ntibacteri  activity of AgNPs was assumed to be relat size, the larger the surface area to volume ratio.  microbial cells. Similarly, Pal et al. (2007)  also shape dependent. AgNPs attack bacterial cel which induce antibacterial effects like denaturati replication and respiratory chain finally leading to death (Fe Sondi, 2004; Morones et al., 2005; Song et al., 2006).  nes et al., 2005), the smaller t facilitates the interaction wi ntibacterial activity of AgNPs i  he release of silver ions  Ne dl  through in the ce  tion against the  a  a  he th  is Is  on of cell membrane, interference with DN  ., 2000; Sondi and Salopek-  ng et  A  Fig. 6: Antibacterial activities of the synthesized AgNPs (150, 100, 50 g/ml) against clinical bacterial isolates (A, Pseudomonas aeruginosa; B, Staphylococcus aureus; C, E. coli; D, Salmonella Typhi).
Antibacterial activities of the synthesized AgNPs at 50-150 ug/ml concentrations are shown in Fig. 6. The particles demonstrated remarkable antibacterial effects against the clinical bacterial strains of mm for biosynthesized AgNPs using leaf strains. Though, the exact mechanism of P aerugin osa, S. aureus, S. Typhi and E. coli by inducing 10-20 nm zone of inhibi test organ sms (Table 1). These results corroborate the antibacterial activities of AgNPs reported in the previous studies (Priyadarshini et al., 2013; Shankar et al., 2014; Salem et al., 2014; Augustine et al., 2014; Lateef et al., 2015a, b; 2016d). For instance, most recently Lateef reported that the cocoa pod husk extract-mediated AgNPs induced zone of inhibition of 10-14 mm against clinical isolates of bacteria while Sale et al. (2016a m et al. (2014) reported zones of inhibition of 7-19 atex of Ficus sycomorus against some bacteri ity of AgNPs is not yet isms. For instance, the a and ibacte researchers have suggested many possible eff rial acti known but ntibacteri activity of AgNPs was assumed to be relat size, the larger the surface area to volume ratio. microbial cells. Similarly, Pal et al. (2007) also shape dependent. AgNPs attack bacterial cel which induce antibacterial effects like denaturati replication and respiratory chain finally leading to death (Fe Sondi, 2004; Morones et al., 2005; Song et al., 2006). nes et al., 2005), the smaller t facilitates the interaction wi ntibacterial activity of AgNPs i he release of silver ions Ne dl through in the ce tion against the a a he th is Is on of cell membrane, interference with DN ., 2000; Sondi and Salopek- ng et A Fig. 6: Antibacterial activities of the synthesized AgNPs (150, 100, 50 g/ml) against clinical bacterial isolates (A, Pseudomonas aeruginosa; B, Staphylococcus aureus; C, E. coli; D, Salmonella Typhi).
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